Conn's syndrome (Primary Hyperaldosteronism) - MADE EASY
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- čas přidán 2. 08. 2013
- Conn's syndrome vs Secondary Hyperaldosteronism
Conn’s syndrome also known as Primary Hyperaldosteronism, is a tumor of adrenal glad which causes excess production of a hormone called aldosterone. The major function of aldosterone is to reabsorb sodium and excrete potassium and acid (H+). Conn’s syndrome can be either a unilateral tumor (located on only one adrenal glad) which are also the most common adrenal tumors or bilateral tumor (located on both adrenal glands). The reason Conn’s syndrome is important is because of the fact that it is potentially curable cause of high blood pressure (hypertension).
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What is Conn's syndrome
What is the most common cause of Conn syndrome?
What symptoms are associated with Conn's syndrome?
How do you test for Conn's syndrome?
Is Conn's Syndrome serious?
Can too much aldosterone cause weight gain?
What is primary hyperaldosteronism?
What is Secondary Hyperaldosteronism?
Conn's syndrome vs Secondary Hyperaldosteronism?
Conn's syndrome and Secondary Hyperaldosteronism?
What is hypertension?
What causes hypertension?
Do I have hypertension?
Causes of hypokalemia?
Great video. Very clearly presented and informative. Just one point to consider regarding the sodium concentration in secondary hyperaldosteronism. Due to congestive heart failure (one of the common causes of secondary hyperaldosteronism), patients often become sodium and water overloaded due to increased renin and thus aldosterone release. The increased renin occurs becuase of reduced effective intravascular volume which is sensed in the kidney as you clearly outline. The reabsorption of sodium and thus water leads to hypervolaemia which we see clinically by a raised JVP and peripheral oedema in some patients. However, angiotensin II also stimulates ADH secretion from the posterior pituitary gland as well as renin secretion from the kidney. ADH acts differently to aldosterone and results in water (but not sodium) reabsorption in the collecting duct. Therefore, although patients with heart failure for example are often sodium overloaded (due to the effect of hyperaldosteronism), the disproportionately greater reabsorption of water means that the patient actually can have low sodium concentration in the extracellular fluid (hyponatremia) due to the dilution effect of increased water intake.
You must be a genius. Very clear explanation. Thanks
Wow, you explained it so concisely-yet so simply as well. Thank you
Fezile Gabellah, How are you doing ? We hope you succeeded very well.
Laid in the hospital after successful surgery. Less than 24 hours since the tumour and gland removed and already there has been significant improvement.
Very clear and concise. Thank you!
Really helps to understand. Thanks.
Really well explained. Thank you and well done.
thank god i clicked on your link. very excited to see what you have to offer. stay fit.
Good vid1
One correction:
s-Na+ will be normal as the Na+ reabsorption will be neutralized by the concomittant H20 reabsorption.
fantastic video, thank you!
Always great lectures, thank you so much
very easy to follow and great explanation thank you
beautifully explained. thank you
love it, thanks a lot for ur time
no one can make it any more easy, thank you :)
fantastic video
explained very well
good job
Thank you so much - wonderful seriously.
Great lecture. Thank you
Great lecture thanks ! :)
Awesome.. Really helpfull thanx a lot :))
thank you so much .very helpful
Great lecture!
Short and simple bro ✌️
Brilliant presentation
you are a good teacher
If u can.post for D.insipidus and SIADH .. Would be great !
Very nice 👍,thanks
perfect explanation tnx
Very nice!
Thank you 🙏🏽
very very thxu i understand your lecture ...........god bless you....
Thanks, very good explanation and simple, dude!
THANKS!
Very nice.
Thank you so much
Thank you
thank you
very good
Hi. Can you explain what happens to the levels of angiotensin 1 and 2 in conns syndrome? Renin is obviously low due to primary hyperaldosteronism. Thanks
excellent
Really your explication is very good. Made Easy is tru
+Edwin Celso Vilca Pajares, Thank you, we're glad you found this video tutorial helpful. Please check out our other videos and feel free to share them with others :)
what abt mineralocorticoid escape phenomenon?
Consider teaching
How can I get your rest videos plzzzzzz???????
what does escape phenomenon mean???
in primary hyperaldosterinoism the Na will be normal dt Aldosterone escape metabolism.
nice one
God bless you
superbbbbbbbbb sir...
Great video, what diet is recommended for patients. I would imagine, DASH diet, low sodium, high potassium. What about water intake. 😊😊😊
I have conn's. Clean diet but spironolactone retains the potassium loss. This or eplerenone is the drug of choice. Alongside controlled bloodwork to monitor potassium levels and kidney function. I nearly died from the potassium loss. I'm lucky.
Tq
How can I get your rest visions plz........?????????????????
I have high Aldosterone (2x-3x normal max value) and severe hypertension BUT normal renin, normal Na+ and normal K+ and no protein in my urine, no cerosis, no adrenal tumors. I've gone to many doctors and they are clueless..:/ would appreciate any ideas as to what I might have.
Maria C check medications! These may be a contributing cause?!
How you doing right now
I know it's been a couple years, but I have the same issues. I've now been on Spiro and clonidine patches for a long while and my BP is under control. How are you doing?
It Is better than CTO
No one
Great video! Thank you!