cAMP - ATP, ADP, cAMP - Adenylate cyclase - Protein Kinase A - G-protein, Cell Signaling - Physio
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- čas přidán 26. 07. 2024
- Cyclic adenosine monophsphate (cyclic AMP) cAMP...alpha blockers, alpha receptors, beta blockers, beta receptors, sympathetic adrenergic receptors...heart rate, stroke volume, baroreceptor reflex, G-proteins. Biology, Physiology and Biochemistry.
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I have epilepsy and miss a lot of my lectures due to seizures, your videos have helped SO much in me being able to learn biochemistry and medicine. Thank you! You make it so entertaining as well
You have brought forth joy and passion that I have been lacking this semester. Thank you.
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You are so funny and you expalin these concepts so well and so clearly! I love this video, thank you sooo much 💖🙌🏼
Of all the videos and articles I read this made the most sense. Thank you for breaking it down, and making it fun with your side comments. Really helped me grasp the concept!
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In the first three minutes i got all my confusion solved, the confusion that has happened after reading a university pamphlet for hours nonsense!
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I've been searching for videos on cAMP, cGMP, and found some very good ones but I wanted to see if you also had a video on this subject, and I found it! Thanks for your effort. Funny, repetitive, really makes me understand better.
I love your videos so much!!! I am preparing for my MCATs and I watch your videos to deeper learn topics. Please keep making more! Thank you for all your help!
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Good luck with your MCAT.
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EXCELLENT resource to put concepts together of what 2 textbook, paid courses and expensive books can offer. After watching your video I feel that I can practically track where the process begins and ends forward and backward. Forever grateful. Thank you
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Your videos are simply amazing. I have a much better understanding of many topics including cAMP!
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I really love this video,that makes sense .
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Thank you so much! I've been stuck confused and didn't want to watch this video coz it's longer than the other cAMP CZcams videos by like 15 minutes. But I was confused even after watching them so I wasted my own time and here I am.
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Nice explanation thank you so so so much
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I am not even studying pharmacology this year, but this was nice
I laughed when you said "it's common sense" 🤣
Thank u for this video and please teach us the biochemistry
ضحى يوسف I have a biochemiistry playlist
❤ love your vibes How to explain and I love your voice more
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Very informative.
Alphones Kuma Thank you so much 😊
just of curiosity are those receptors receiving information outside the membrane which because of the signal they RECEIVE, the START the process of producing the cAMP? , are they the G protein couple receptors?
Is there a place i can get the transcript of this lecture from ? 18 minutes video made me understand so much about antiplatlets therapy, and some of the most common shock hf interventions we take ,, and even aminophyline toxicity .. thank u man
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I noticed a mistake at 13:05. You say Ca+ channel blockers 𝐢𝐧𝐜𝐫𝐞𝐚𝐬𝐞 cardiac contractility, however, the drawing states that it 𝐝𝐞𝐜𝐫𝐞𝐚𝐬𝐞𝐬 (this is correct).
They decrease contractility.
Thank you so much for correcting me.
@@MedicosisPerfectionalis I have a question! I'm not very clear on the cAMP effects on Angiotensin? Doesn't it inhibit this process (therefore, causing vasodilation)? I've tried watching that part a few times but I don't understand. Please, enlighten me!
Does cAMP cause both vasoconstriction and vasodilation? Beta-agonists increase cAMP, therefore causing vasodilation. But Beta-blockers cause vasodilation by decreasing cAMP because of the A-T pathway? Why doesn't cAMP *only* cause dilation/relaxation in smooth muscle?
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Really helpful
Muhammad Uwais Thanks 😊
@@MedicosisPerfectionalis if u don't mind. Can you share about muscle reflex?
A video?
@@MedicosisPerfectionalis yes
Muhammad Uwais like the knee jerk reflex, ankle reflex,...etc?
can you make video on cGMP mediated vasodilator
this is so cool. hahha thanks for yhe good pneumonic 😎
My pleasure 😇
if you use b-blocker, which means b1 receptor is not working, so no stimulation of Gs, so no cAMP. no cAMP means contractility is decrase and vasoCONSTRICTION, no????
means b-blocker can cause vasoconstriction leads to increase BP
please correct me if i misunderstood
This easier to understand then my teacher 💀
PDEs are also of different types...There is PDE1,2,3,4.Could u shed some light on the location?..are they all present in the cytoplasm?
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Haha 😂
Frodo Beutlin Welcome to the family!
cAMP acts on smooth muscle of blood vessels and cause vasodilation and a decrease in bp. On the other hand it increase angiotensin ii and aldosterone and causes vasoconstriction and salt and water retention and increase bp. So which is the predominant effect?
Please help me out.
did you find an answer to this? I'm wondering the same thing
Man you are asm just awsm
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How cAMP contract heart muscle and dilate smooth muscle??
Super
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Hello ! Anyone can explain me why a2 receptor -> Gi pathway -> low c AMP, but it cause relaxation?
(Source: b. α2 Receptors
are located on sympathetic postganglionic nerve terminals (autoreceptors), platelets, fat cells, and the walls of the GI tract (heteroreceptors).
often produce inhibition (e.g., relaxation or dilation).
Mechanism of action: Gi protein, inhibition of adenylate cyclase and
decrease in cyclic adenosine monophosphate (cAMP). )
great
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