Really enjoying your videos! I have a useful mneumonic for the causes of ST elevation, it is ST-ELEVATION: S: Syndrome (brugada) T: Takotsubo E:Electrolytes (hyperkalemia) L: LBBB E: Early repolarization V:Ventricular hypertrophy A: Aneurysm T:Treatment (pericardiocentesis) I: Injury ( Acute MI, cardiac contusion) O: Osborne waves ( hypothermia) N: non-occlusive vasospasm.
That's for making this video. I had a pt with c/p. J point elevation, Tall, broad based concave t waves. 12 lead showed concave hyper acute t waves as you showed in the video. I was told no MI! Like you said in your video (paraphrasing) do not blow off concave t waves as a normal, look at the patient/history and the complaint. Trop came back >1000. T waves improved with Nitro gtt. SUBSCRIBED!
Can you show an example calculation of a QRS-T angle? As an aside, this is the absolute best series describing EKG's that I've come across. I appreciate how you break down concepts and vectors to their base level, giving students like a solid framework of which to work. I appreciate you Dr. Strong!!
thank you dr eric. i would like to ask about 3:32 isn't during lbbb. deep negative deflection on v1-v3 considered DEEP S WAVE? why in this video you considered them as pathological q wave?
Hi Dr. Eric! Thank you so much for the great content! Greetings from Germany! I guess I'm being very picky, but in your video on bundle branch block you used the same EKG for LBBB and explained that we can see deep S waves in V1 with secondary upsloping ST elevations and prominent T waves.. here you call the S waves Q waves instead.. wouldn't it technically be a QS complex due to the lack of any upward deflections here?
dr strong - do you have powerpoint files that accompany your lectures? that helps for review. sorry, i know i'm asking a lot. if not, i'll just screenshot key parts. these videos are better than any textbook!
What do you believe are the possible causes for the delayed and absent R wave progressions in 7:16 in and 7:56 respectively, assuming the precordial electrodes have been placed correctly? I know that both the limb leads and the clinical context are missing, but I am really curious to see what people would consider with this image alone.
Why isn't an old MI an etiology of low voltage? If the myocardium is replaced by scar tissue, wouldn't a severe MI lead to low voltage in some cases? Also, I am confused about when you first show a "normal" q-wave in V1 at (2:15) and then explain that a q-wave in V1 is considered pathologic. I didn't hear you explain this, but the EKG-strip above the axial section of the heart says "normal". What's the deal? Thanks again for these terrific videos!
Hello, i love your video and explanation. but I have a question. Is STEMI a diagnosis, or just EKG interpretation? If STEMI is a diagnosis, then how about AMI (Acute Myocard Infarction)? Please kindly answer my question. Thank you.
STEMI is a diagnosis requiring both EKG criteria as well as the overall clinical picture. "Acute myocardial infarction" is not a particularly common formal term in clinical medicine in the US per se, but refers to the combination of STEMI and non-STEMI (the latter of which is just an acute MI in a patient which doesn't meet the EKG criteria for a STEMI). More common, at least in the US, is the term "acute coronary syndrome", which includes STEMI, non-STEMI, and unstable angina.
So, if someone just give me an EKG sheet of a patient showing ST Elevation on inferior and anterior lead, we couldn't say it's STEMI yet, right? Or we could already say it's STEMI regardless the clinical picture or the cardiac enzyme? (which is not checked yet). Thank you so much for responding. It helps me understand cardiology better.
Dear dr. Strong, in your opinion, what is the cause/clinical significance of QRS notching that can sometimes be seen in one or more frontal leads when there is no RBBB? I occasionally see this in otherwhise healthy individuals. Thank you for another great video
+Albin Mammen QT prolongation is briefly mentioned in the Systematic Approach video (czcams.com/video/ENyBhCJ2llY/video.html&nohtml5=False) at ~18:40. I'm intending to have QT prolongation actually be the subject of its own video (including the fascinating genetics and phenotypes of congenital QT prolongation), but just haven't gotten to it yet. Too many other topics (and my day job...)
Really enjoying your videos! I have a useful mneumonic for the causes of ST elevation, it is ST-ELEVATION:
S: Syndrome (brugada)
T: Takotsubo
E:Electrolytes (hyperkalemia)
L: LBBB
E: Early repolarization
V:Ventricular hypertrophy
A: Aneurysm
T:Treatment (pericardiocentesis)
I: Injury ( Acute MI, cardiac contusion)
O: Osborne waves ( hypothermia)
N: non-occlusive vasospasm.
What about pericarditis?
Sorry, but just to comfirm... There are several other things that can look like a heart attack? (ie. St-elevation).
@@chrism6904 yes
That's for making this video. I had a pt with c/p. J point elevation, Tall, broad based concave t waves. 12 lead showed concave hyper acute t waves as you showed in the video. I was told no MI! Like you said in your video (paraphrasing) do not blow off concave t waves as a normal, look at the patient/history and the complaint. Trop came back >1000. T waves improved with Nitro gtt. SUBSCRIBED!
Can you show an example calculation of a QRS-T angle?
As an aside, this is the absolute best series describing EKG's that I've come across. I appreciate how you break down concepts and vectors to their base level, giving students like a solid framework of which to work. I appreciate you Dr. Strong!!
Thank you Dr Eric Strong.
Excellent work dr Strong
Explained nicely... MAA Shaa Allah
Thank you so much Dr.
Thank you very much Dr Eric Strong
thank you dr eric. i would like to ask about 3:32
isn't during lbbb. deep negative deflection on v1-v3 considered DEEP S WAVE? why in this video you considered them as pathological q wave?
Very well made videos! Thank you for your effort!
Hi Dr. Eric! Thank you so much for the great content! Greetings from Germany!
I guess I'm being very picky, but in your video on bundle branch block you used the same EKG for LBBB and explained that we can see deep S waves in V1 with secondary upsloping ST elevations and prominent T waves.. here you call the S waves Q waves instead.. wouldn't it technically be a QS complex due to the lack of any upward deflections here?
Nice work....تم التحميل
Thank you Dr Strong.
dr strong - do you have powerpoint files that accompany your lectures? that helps for review. sorry, i know i'm asking a lot. if not, i'll just screenshot key parts. these videos are better than any textbook!
What do you believe are the possible causes for the delayed and absent R wave progressions in 7:16 in and 7:56 respectively, assuming the precordial electrodes have been placed correctly? I know that both the limb leads and the clinical context are missing, but I am really curious to see what people would consider with this image alone.
Great videos, extremely educational. Keep up the good work and thanks!
I believe you have LAFB and LPFB q waves switched!
Great video
Why isn't an old MI an etiology of low voltage? If the myocardium is replaced by scar tissue, wouldn't a severe MI lead to low voltage in some cases? Also, I am confused about when you first show a "normal" q-wave in V1 at (2:15) and then explain that a q-wave in V1 is considered pathologic. I didn't hear you explain this, but the EKG-strip above the axial section of the heart says "normal". What's the deal? Thanks again for these terrific videos!
thank you Dr Eric. I would like to ask if myocarditis can be an etiology for low voltage (beside ST elevation) ?
Yes, myocarditis can absolutely cause low voltage. It was an unfortunate omission on my part!
@@StrongMed thank you so much respected teacher for your scientific objectivity and for being humble
What is the reason for S wave in lead V6?
Hello, i love your video and explanation. but I have a question. Is STEMI a diagnosis, or just EKG interpretation? If STEMI is a diagnosis, then how about AMI (Acute Myocard Infarction)? Please kindly answer my question. Thank you.
STEMI is a diagnosis requiring both EKG criteria as well as the overall clinical picture. "Acute myocardial infarction" is not a particularly common formal term in clinical medicine in the US per se, but refers to the combination of STEMI and non-STEMI (the latter of which is just an acute MI in a patient which doesn't meet the EKG criteria for a STEMI). More common, at least in the US, is the term "acute coronary syndrome", which includes STEMI, non-STEMI, and unstable angina.
So, if someone just give me an EKG sheet of a patient showing ST Elevation on inferior and anterior lead, we couldn't say it's STEMI yet, right? Or we could already say it's STEMI regardless the clinical picture or the cardiac enzyme? (which is not checked yet). Thank you so much for responding. It helps me understand cardiology better.
Est-ce que je peux avoir la vidéo en français merci
Can you help me with book for reading ECG easily with much abnormalities
IMHO, the best introductory ECG text is "The Only EKG Book You'll Ever Need" by Thaler.
Dear dr. Strong, in your opinion, what is the cause/clinical significance of QRS notching that can sometimes be seen in one or more frontal leads when there is no RBBB? I occasionally see this in otherwhise healthy individuals. Thank you for another great video
Ur videos give me headache 🤕
They r difficult 😞
do you mention QT prolongation anywhere in your videos?
+Albin Mammen QT prolongation is briefly mentioned in the Systematic Approach video (czcams.com/video/ENyBhCJ2llY/video.html&nohtml5=False) at ~18:40. I'm intending to have QT prolongation actually be the subject of its own video (including the fascinating genetics and phenotypes of congenital QT prolongation), but just haven't gotten to it yet. Too many other topics (and my day job...)
Hi Dr.Eric,
What is the name of your Facebook page if you have one?
Thank you
I'm sorry, but I don't use Facebook- only Twitter.