Aortic stenosis / Aortic valve disease : Pathophysiology Usmle step 1
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Aortic stenosis / Aortic valve disease : Pathophysiology Usmle step 1
Aortic stenosis is narrowing of the aortic valve due to calcification of the valve leaflets or valvular damage.
Aortic stenosis commonly occurs at an early age in patients with congenital bicuspid aortic valves.
The most common cause of aortic stenosis is calcification of the valve leaflets with age, with the prevalence of aortic stenosis increasing rapidly beginning in the 7th decade.
Aortic stenosis can also result from rheumatic fever. Though rheumatic fever is most strongly associated with mitral stenosis, other valves may also be affected.
Aortic stenosis is classically associated with syncope, chest pain and dyspnea on exertion. Aortic stenosis can progress rapidly, leading to sudden cardiac death.
Aortic stenosis can lead to left ventricular hypertrophy as the myocardium contracts against the narrowed valve opening. The myocardium outgrows its blood supply, leading to ischemia, progressively worsening chest pain, and dyspnea from pulmonary congestion.
Since aortic stenosis leads to myocardial ischemia and a fixed cardiac output, symptoms initially present during exercise. As the disease progresses, however, symptoms begin to occur at rest.
Aortic stenosis causes a pansystolic crescendo-decrescendo murmur heard loudest in the second intercostal space at the right sternal border.
The murmur often radiates to the carotid arteries.
The murmur decreases in intensity with decreased preload (such as in the Valsalva maneuver).
Aortic stenosis is associated with an S4 heart sound as well as “pulsus parvus et tardus,” or peripheral pulses that occur weak and late relative to the heartbeat, due to the slow emptying of left ventricle to the systemic circulation.
CXR shows left ventricular hypertrophy.
Echocardiography shows a narrowed valve area with increased transvalvular pressure gradient. Other findings may include left atrial enlargement and left bundle branch block. The most accurate way to quantify the transvalvular pressure gradient is with cardiac catheterization. aortic stenosis pathology
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It's the best explanation I have found about this topic, thank you so much, doctor.
Thank you Doctor for a very thorough and easy to understand presentation.
I am having TAVR done tomorrow on 5-3-21. This helps me understand more of what is going on. My gradient is 72, the opening of the AV is.6 the EF is 40%.
I have an aortic aneurysm that is 4.3. I am aasymptomatic but passed out 3 months ago and 3 years ago.
Mostly I feel good but do get out of breath when I exert myself.
Because of presentations like yours I look forward to gettin TAVR and getting back to normal.
I THANK YOU.
Glad it was helpful!
Very helpful sir...thank you
very good video for usmle step 1
ME REALLY THANKFUL TO U SIR
GOOD PRESENTATION 👍 that made to Concrete in My mind.
Thank You 😊👍
Nice to hear that ... ur most welcome ...keep watching
very good explanation. realy thant u so much doctor
good video sir
Best concept clear sir
thank you so much
Very good explanation of AS will be watching more in the future
Tysm
Thank you so much . So wonderfully explained .
Ur most welcome , thanks for watching
I have border line Left Ventricle Hypertrophy . Dr York on you tube in England taught that we should find out the reason. . You have brilliant explanation for a non cardiologist to understands and Ultra sound reports mild Aortic calcification.Now I can speak to my Doctor with a little bit of understanding , Again Your an excellent teacher .
Very good explanation thank you doctor....
Glad you liked it
Thank.you.so.much.sir.for.your.informative.lecture.
Ur most welcome
Simply excellent. Very grateful for clear, concise and well presented video. Thank you for the great channel. 8/8/2018 😊
Thank u so much
Crystal clear.
Thank u
Very well explained 👍
Thank you
Thank you, Sir!!!
So, Sir SBP is high along with the pulse pressure???
S Bro SBP high wid Pulse Pressure
Speechless.
🙏🙏🙏
thank you so much .its is a life saver ]🙏🙏
You're welcome!
Aortic stenosis calcified and thickned biscusipid need surgery.
Aortic flow:4.5m/s
Gradient ppg70mmhg
Mean 40mmhg.
LVEF60%
Good LV function.
Excellent 🙏
Thanks a lot
kindly would u explain rheumatic heart disease
RVH main ' a' wave prolonged hoti h to yaha left ventricular hypertrophy mai kaise ho rhi ?
شكرا لكم
خل يوجد له تدبير علاجي غير جراحي
Thank you
Is there
Any curing drugs..pleas
🙌🏽🙌🏽🙌🏽
Awesome ❤
Thank you! Cheers!
super bhanu
thank u
Mind penetrating explanation 👌👌👌
Glad you liked it
I understand syncope and angina, but I do not understand dyspnea due to AS.
Dyspnea due to pulmonary edema
Dr.G.Bhanu Prakash thanks for reply pulmonary edema is due to increased LA pressure which causes blood to be backed up in the lungs? Thanks sir it is clear now
Sir, please amke videos on murmur
make*
Sir.. Please explain.. That, HOW A WAVE BECAME PROMINENT due to LEFT atrium enlargement 😑😑
A wave in jvp means left atrium . A means atrium . Therefore if atrium is big a wave will become big .
Would you plz do the finding in echo in valves disease special AR ,MR,MS,MR
The changes i mean in echo which one will be dilated Lv , which one will be Thicked Lv ,ect
Plz never understand standard
❤❤❤
Thank u so much
Ventriculaaaaaar
Is aortic stenosis the same thing as ventricular stenosis?
noo terminology is different as stenosis might be at septal level also
Sir ..well explained.
Want to.contact you regarding my condition .
Can i get your email
The member ship in India ruppee