Brain Stem Lesion Mnemonic - CRASH! Medical Review Series
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- čas přidán 6. 08. 2024
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ADDENDUM: You should also be familiar with the basilar artery infarction, which causes "locked-in" syndrome!
(Disclaimer: The medical information contained herein is intended for physician medical licensing exam review purposes only, and are not intended for diagnosis of any illness. If you think you may be suffering from any medical condition, you should consult your physician or seek immediate medical attention.)
Medical schools around the world need to pull their socks up and provide benchmark lectures to their students such as this one.
This is pure genius! Why is this diagram missing in our books! Awesome doctor,. Awesome explanation!
It’s been so long since you uploaded. Thank you so much for all the hard work Paul. Forever grateful. :D
Thank you for sharing all of your videos. They are great and I am deeply thankful for them and the work you've done to make them.
These lectures are a treasure trove of information which a family doctor should know.
Wish there was an easy way to send money to him.
This comes exactly 1 week after my proffs. I looked for videos and couldn’t find it. I will make sure my
juniors benefit from it
Paul bolin we missed you!
Glad to hear from you again!
I missed you so much! Thanks for sharing your knowledge once again!
These courses are good enough for any medical board exams through the world. So doctors please help your own countrymen and relatives because there are extreme shortage of doctors. Doctors are well paid and highly respected in the communities of your motherland.
Thank you . Great presentation.
Lovely presentation . Well delivered
Thank you very much! This has helped me a lot ❤️
So nice to hear from you. Thanking the Lord that He has kept you safe. Welcome back Dr. Bolin.
Paul bolin is back..... Thank you so muchhhhhhh
Awesome simplification sir
Missed you man! Thanks so much for all the awesome content on your channel! It has been so helpful!!!!!
This is a great video - thanks
Phenomenal explanation.
Wow completely life changing video! Thank you so much just thank you!!!
Thanks a lot Dr
Thanks alot for sharing g8 video.
Its quite informative.
Keep it up .
Its really very easy to understand and quite interesting.
❤️❤️ loved this simplified version... So easy to remember... I hv wasted so much of time trying to mug them
Thank you 🙏🏻
Amazing video
Thank u so much doc we missed u
nice to hear from you
THANK YOU
Omg you're back!!! Yay
Best explanation ever!
Thank you soo much
What an amazing video!
Woahhhh welcome back 🙏🏼
I missed your lectures, hello from the Philippines 🙂
THIS IS GONNA SAVE ME SO MUCH TIME. Thank you so much for this wisdom!
Great video❤
Excellent
this is just beautiful
Welcome back
Back after a long time...
Good to see you.
So will you upload regularly now?
Thursday September 29, 2022. Neurology: Brain Stem Lesions (Midbrain [CN III, CN IV], Pons [CN V, CN VI, CN VII, CN VIII], and Medulla Oblongata [CN IX, CN X, CN XII]), Central Nervous System. Medial Cranial Nerve Anatomy follows: Oculomotor Nerve (CN III), Trochlear Nerve (CN IV), Abducent Nerve (CN VI), Hypoglossal Nerve (CN XII), while Lateral Nerves are Pathophysiology of Ischemia/Infarction and/or Compression: 1) Medial Medullary Syndrome (Anterior Spinal Artery [ASA]); 2) Lateral Medullary Syndrome (Wallenberg Syndrome) involves the Posterior Inferior Cerebellar Artery (PICA) and manifests as a 1) Loss of Gag Reflex (Mediated by Glossopharyngeal and Motor Vagus Nerve), 2) Vertigo, Vomiting Nystagmus (Vestibulocochlear Nerve), 3) Anesthesia due to CN V or Trigeminal Nerve, 4) and Chewing Difficulty (CN V2). Also, 5) Sympathetic Pathway is involved and manifests with Horner's Syndrome (Miosis, Ptosis, and Anhydrosis) along with 6) Contralateral Loss of Pain and Temperature Sensation (Spinothalamic Tract Involvement); 3) Lateral Pontine Syndrome implicates the Anterior Inferior Cerebellar Artery (AICA) and is Stereotyped by 1) Facial Drooping Symptoms and 2) Ataxia (Facial Nerve Origin), 3) CN VIII Symptoms: Vertigo, Vomiting and Nystagmus, 4) Facial Paralysis and Dysphagia (Cranial Nerve V, the Facial Nerve Involvement) ; 4) Medial Pontine Syndrome involves the Basilar Artery; 5) Weber Syndrome (Superior Alternating Hemiplegia) has the Paramedian Branches of the Posterior Cerebral Artery (PCA) will present with 1) Oculomotor Palsy (Down and Out Eye Morphology), 2) Upper Motor Neuron Paralysis (Contralateral Spastic Paralysis: Hypertonia, Positive Babinski Sign), 3) Contralateral Parkinsonism (Bradykinesia, Rigidity) due to Substantia Nigra (SN) Involvement; and the 6) Dorsal Midbrain Syndrome is Compression/Injury of the Rostral Interstitial Nucleus of Medial Longitudinal Fasciculus (riMLF), Structure Perfused by the Basilar Artery (Parinaud Syndrome) and Presenting with Upward Palsy or Best regarded as a Paresis; Aetiology is Compression; and 7) Cerebellopontine Angle Syndrome (CPAS) is Compression of the CN V (Ipsilateral Masseter/Temporalis, Medial Pterygoid and Lateral Pterygoid Muscle Weakness, Loss Of Corneal Reflex) and CN VIII (Acoustic Neuroma/Meningioma) showing 1) Tinnitus and 2) Hearing Loss; Ataxia (Cerebellum Involvement). A Non-Communicating Hydrocephalus is also Present in CPAS due to Fourth Ventricle Obstruction. In short, Cranial Nerve Involvement will hint as to Ischemia/Infarction/Compression Location while Motor and Sensory Deficits (Signs and Symptoms) will hint the Particular Involved Vessel. Goodness, Clinicopathological Correlation is indeed the Essence of Medicine. MD Paul W. Bolin, die Krankheit macht die Gesundheit aber ich manchmal dachte Gesundheit macht die Krankheit. Ich nur bin einer Mann. Prost!
Anterior spinal artery doesn't give off Pica branch it comes from vertebrae directly
Good to see you
this is a really good mnemonic
this is gold
Welcommeeee backkkk
amazing
Could it be? Dr. Bolin back at it again?
7 years on this website wow
Great video, it helped me a lot but there is one thing that is not explained. What happens to the DCML for weber's syndrome because for medial medullary syndrome you mention loss of fine touch proprioception and vibration contralaterally but in Weber's syndrome you don't mention anything about DCML. Doesn't DCML run through there so it is affected or am I missing something. Other than that great video and you earned a new subscriber 👍.
My wife has brain lesions in her brain stem and cerebellum, 12 nuero docs and 2 yrs later noone has given her a concrete answer, any studies I can look at to help her understand them?
Sir, I was looking for neurocutaneous diseases on hour channel? Please tell me which lecture would contain this.
Conceptual one ☝
Thank you very much sir. I am a 2nd semester student. I would like to pay once i reach the 5th semester. Thank you very very much
better than boards and beyond's rule of 4s video
No estará en español 😣?
Dr.paul in previous lectures in ASA stroke you said that ASA supply STT and CST , so the dcml will not be affected in ASA stroke
here you say that in ASA stroke the DCMl and CST will be affected
You said that DCML supplied by PSA
amazing ，
Missed you.
misss u
Good nice
6:55 I think you mean factors of 12. Yes, I'm knit-picking 😁
Hi, at 18:09 I think there's a small mistake. In lower motor neuron lesion, entire facial nucleus gone, so it should be entire ipsilateral face deficit, right? Please look into and confirm. Otherwise, this is amazing video, thank you!
I had the same concern, I think he mixed that up. if the facial NUCLEUS is knocked out it should only be the ipsilateral lower face that is not innervated, and the forehead will have innervation from the contralateral side. if the facial NERVE is knocked out it will be the entirety of the ipsilateral face that will not be innervated.
Ai winkunzi kakhulu mbhemu yazi wavele wayenza yasobala okwezinqe zesele💥🤝💪
To be specific, tuberous sclerosis
can we get the slides ?
👏
yaaay !!
😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍😍This is so good
I love you
I wanna marry you
wallah
wallahahahahah
NOOO HARAM