Cell Adhesion Molecules Part 1

In this video we discuss the four different families of cell adhesion molecules, specifically looking at the immunoglobulin superfamily of cell adhesion molecules.

[@kylep8941]

Good luck to all you fellow MCAT studiers that are here because of the MilesDown deck. You're doing great!

[@andrewnorato9924]

I have watched many of your videos. You are an excellent instructor! Your videos are an excellent supplement to my (very confusing) textbook! Thank you!

[@internationaljournalofpath5931]

easy, simple, vivid explanation. Thanks. And thanks to the God, most merciful, the most wise and all powerful for creation of all these useful, essential molecules with utmost wisdom!

[@uhuihiuihiuh3042]

They were formed through evolution, not created by some invisible sky god

[@m.shyamganeesh7421]

pproust how can you create evolution ? It’s not a physical thing , don’t ask dumb questions .

[@mrwho156]

M.Shyam Ganeesh the evolution must’ve started by one creation at least right? Who created that to start with?

[@jamesharwood2937]

Thanks for this video. I know nothing about this area of science but needed to get a basic understanding of what intergrins were about. Excellent presentation by someone who cares about others. Thanks.

[@elliotnicholson5117]

Thank you for watching and writing such a nice comment,

[@carolinegreen4771]

The recruitment of leukocytes from the blood stream to extravascular sites of inflammation is necessary for an effective inflammatory response. The recruitment is mediated by #Cell #Adhesion #Molecules, cytokines and chemoattractants which together coordinate this complex series of events. The adhesion cascade involves five steps which occur in sequence but may overlap in time. At sites of inflammation in the systemic circulation (as opposed to the pulmonary circulation), post-capillary venule endothelium becomes activated by the local generation of a variety of inflammatory mediators. Histamine, thrombin and LTC4 lead to a rapid expression of P-selectin on the endothelial surface while the early proinflammatory cytokines such as IL-1 and TNFα lead to de novo synthesis of both P- and E-selectin as well as ICAM-1 and VCAM-1 resulting in increased surface expression at time points greater than 2 hours following stimulation. When endothelium is exposed to these cytokines especially in the presence of FN for prolonged periods (as during chronic infection), the volume of the endothelial cell expands and it becomes columnar, similar to the morphology in high endothelial venules in lymph nodes.
Creative Biomart

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You are great!

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Thank you

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I need this in a PDF file lol

[@hifzarai5868]

Thank you!

[@ummewaseem5983]

Thank you!

[@TheVuze]

Okey

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If u r asking about the black pen,,,,, well it is (uni-ball eye...) Mitsubishi pencil ( made in Japan ) 😁✌🏻️

[@ElizabethRey97]

Uniball

[@saikumar802]

LAD 1 | LAD2
______________________________________________________
AR AR
Deficiency of B 2 chain Def of sialyl-lewis x*
of B 2 integrin Rolling is impaired*
C/F-
Delayed sep of cord stump* Bombay blood gr*
leukocytosis* Leukocytosis*

[@xender6969]

Please cut out the noise in audio

[@hudaal-khalidi2558]

I REALLY HATE THIS SUBJECT