Differentiating shock | Circulatory System and Disease | NCLEX-RN | Khan Academy
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- čas přidán 23. 11. 2014
- Created by Ian Mannarino.
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Great summary clear and very concise. Thank you.
Thank you Doctor khan 🙏
Thanks so very much - best videos out there!!!
Another parameter to evaluate and that would help to distinguish the several kinds of shock would be the skin temperature and sweat. Still very good video, congrats!
sorry I have a confusion here, why SmvO2 has to be low in dissociative shock? .. the pathophysiology in dissociative shock involves impair in the delivery of oxygen by hemoglobin to the tissue, doesn't that mean that the oxygen never reach the tissue in sufficient amount? and also that there are a lot more 02 returning to heart than it reaching the tissues ... so that SmvO2 level would be high ... pliz clarify my doubt ... thanking you ..
Chinneichong Haokip m thinking the same
I'm not sure, but I think the issue is in measurement. MVO2 measures O2 that isn't binded to Hb. But it's only my guess
same question..
Dr. Mannarino posted the same video on his own channel and responded to somebody with this comment:
"Hi John, that's an excellent question! You are in fact correct, the MVO2 is high, since oxygen remains bound to hemoglobin. What I meant to say is that the oxygen content in the blood that is NOT bound to hemoglobin is low, because that will be pulled out by the tissues. Thank you for pointing this out."
All of my nursing textbooks say that you will have a decreased cardiac output for Anaphylactic shock...???
You are correct according the most recent research. The idea of how anaphylaxis caused shock (vasodilation, decreased svr) was actually not well understood until recent studies in the past few years, which found that the problem seem to actually be due to decreased CO and actually not a decrease in SVR.. google scholar will give you the studies easily
Metabolic Theory of Septic Shock
Please do a search for the above
Core tip: For decades septic shock has been attributed to an over-active immune response. However, immune modulation has failed to reduce mortality, casting doubt on a direct causal role for the immune response in the development of septic shock. A closer look suggests that septic shock is the result of a generalized build-up of hydrogen peroxide, a toxic cellular by-product generated as a consequence of the hypermetabolic state that accompanies a systemic immune response. This finding points to the systemic accumulation of hydrogen peroxide as a significant risk factor for the development of septic and non-septic shock syndromes.
Agreed. There are many parameters here that are off if you look in the medical texts. Especially when he discusses PCWP, many of this do not match what the medical texts are teaching. Be careful.