Visual Pathway & its defects - Optic nerve - Optic Chiasm - Optic tract - Lateral Geniculate Body
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- čas přidán 7. 10. 2023
- Visual Pathway & its defects - Optic nerve - Optic Chiasm - Optic tract - Lateral Geniculate Body in the metathalamus of the thalamus - optic radiation (including Meyer’s loop in the temporal lobe) - primary visual cortex in the occipital lobe of the cerebral cortex (cerebrum)…Visual field defects can cause complete blindness, bitemporal hemianopia (pituitary adenoma or pituitary tumor), contralateral homonymous hemianopia (with or without macular sparing)، quadrantanopsia,…etc. Neuroanatomy playlist.
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23:35, 10 and 11 are altitudinal visual field defects
Causes can be
G: glaucoma
O:optic disc drusen
A: ant ischemic optic neuropathy.
Thank you for the video. please correct 16:51-53 it should be PARIETAL not temporal
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I don't know that much but i have very random question can we directly connect eye to optic chiasm
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My professor just read slides for us
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Please tell us what is the answer to the second question? I have a neurology exam next week 😢😢😢. Excellent explanation thank you very much.
Love the 1984 reference!
Broadmann area 17 is in the visual cortex.
18 and 19 are visual association areas.
Excellent!
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Nerve is tract; Truth is lies.
Amazing 😊
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Can anyone explain me what did he mean by ( the macula has bilateral cortical representation even if one cortex or one half or one hemisphere is “screwed” the macula is “spared” because the macula “get also supply” from the other cortex)
I don’t speak English well but I understood everything at the beginning except this, also this bilateral cortical representation what is it exactly ?
Thanks 🙏
It means that sensory afferent fibers from each macula will reach both cerebral cortices.
Broadman area 17
What happens in dyslexia
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Is the 11th defect because the temporal optic radiation of both right and left cerebral hemisphere has a lesion?
But why macular sparing?🤔
بنها فخورة
22:37 If the macula has a bilateral cortical representation why wouldn’t the contralateral cortex compensate for the heminopia in a unilateral optic tract, LGB or radiation lesion?
Because when there’s a lesion in the optic tract, LGB, or the entire optic radiation, the macula doesn’t have any path to communicate with the cortex at all. Each macula connects to both cerebral cortices through nerve fibers, but these nerve fibers have to travel through optic nerve, optic tract, LGB, and optic radiation. If you injure any of these areas, then the only way the macula has to reach both occipital cortices has been severed!
Does that make sense?
@@MedicosisPerfectionalis my question is if the lesion occurred after the optic chiasma, won’t the nasal fibers have already crossed to the other side to reach the contralateral cortex carrying macular visual impulses as well?
16:51 small mistake here, upper lobe is parietal lobe not temporal lobe.
He said the upper is parietal and lower is temporal.... not sure how you heard him differently
Broadmann area 17
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and for the quiz ...
could u check my answers..
question.(10) first part
bilateral inferior quadrantanopia w\sparing macula
second part > in the parietal optic radiation both sides
question(11) first part
bilateral superior quadrantanopia w\sparing macula
second part > in the temporal optic radiation both sides
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another goated video
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17??
it actually is area 17, gg
First comment ❤
Thank you!
Please who understood the joke at the beginning 😅
17 1:52
Broadmann area 28