![Janine LeBlanc-Straceski](/img/default-banner.jpg)
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Janine LeBlanc-Straceski
United States
Registrace 9. 07. 2014
Molecular Biology, Genetics, Developmental Biology
Video
Mitochondria Lab Graphing Tutorial Video
zhlédnutí 156Před 6 lety
For Google Sheets. How to calculate Average and Standard Deviation. How to create a bar graph with error bars corresponding to the StdDev.
OSMOSIS LAB Chart Instructions 720p
zhlédnutí 1,1KPřed 6 lety
This video shows how to make a a basic chart or graph from data in a google sheets file.
OSMOSIS LAB Formulas in Table Instructions 720p
zhlédnutí 539Před 6 lety
This tutorial shows how to use use Google Sheets to record raw data and calculate % change in weight and volume.
Clyclosis in Elodea 100x magnification
zhlédnutí 1,3KPřed 6 lety
Clyclosis in Elodea 100x magnification
Unit 14C Transduction and Transformation
zhlédnutí 580Před 8 lety
Unit 14C Transduction and Transformation
Unit 14A Bacterial Genetics Conjugation
zhlédnutí 891Před 8 lety
Unit 14A Bacterial Genetics Conjugation
Unit 13C Chromosome Number and Structure
zhlédnutí 502Před 8 lety
Unit 13C Chromosome Number and Structure
Unit 11B Variations on Mendelian Inheritance
zhlédnutí 454Před 8 lety
Unit 11B Variations on Mendelian Inheritance
Unit 11A Sex Determination and Sex Linkage
zhlédnutí 892Před 8 lety
Unit 11A Sex Determination and Sex Linkage
Unit7G DNA Mutation and Repair Triplet Repeat Expansion
zhlédnutí 1,3KPřed 8 lety
Unit7G DNA Mutation and Repair Triplet Repeat Expansion
In which book , are these lectures ?
Thank you so much for this explanation. It was very straight-forward and helpful! Please keep making videos!
wow thank u
Glad it helped.
There is not much talk about the importance of healthy motor molecules to help with Mitosis, Meiosis and Mendel. Like the signal switch to turn on by a motor molecule of Mitosis
Correct. That is an active research topic for the cell biology of mitosis, meiosis, all of the special cases (such as asymmetrical cytokinesis) and all the ways it can go wrong (as in non-disjunction).
@@janineleblanc-straceski3181 i love to talk to you more if possible about such a subject. i have no degree i cant afford college yet. I just a deep passion for helping Cancer patients that dose more good than bad some day. so the health of the motor molecules leans alot on the ATP for them to get around and Cancer has alot of factors one is it is a uncontrolled situation which is connected to the Mitochondria they activate caspase which helps with Apoptosis . so in short test the health of the motor molecules mainly the Kineasen and Dynien can be tested because the motility of the mitochondria is as far as my studies show done only by them. There is more i will see if i can chat with you more first
@@joshuabowman7210 Since you do have access to a computer, try this: www.edx.org/
@@janineleblanc-straceski3181 thanks
Very interesting statement saying Viruses can transfer genetic material from one bacteria to another i usually thought viruses try to take control of there own RNA to spread there genitic material ? Can viruses transfer of Mitochondria DNA one to another ?
The only video that helped. Thanks a lot. :)
I’m glad you found it helpful!
Thank you. It was very nice talk and a good summary about BRCA1, BRCA2
There is an omission around 6:12. In the transition to the next slide I neglected to emphasize that the table contains data from the F2 generation - the offspring of the mating between the heterozygous female fly and the hemizygous male with all three recessive mutant alleles.
I can't thank you enough for these videos!!!!!!!
I very glad they are helpful.
This is quality content!
Thank you so much. I've been thinking of a way to find everything related to this topic in just one video. Thank you.
Thank you so much for this video! It helped me soooo much.
Glad you found it helpful.
Where's your website?
Well explained and nice slides
Can you tell me which books should i use for bacterial genetics
Can you please tell us how all histone intract to each other in octamer and how h1 is bound
I have no words to thanks you You are save my life
i seriously wish you were my professor
Well, I guess I am! Good luck!
Wow all of what i can say all the respect to you you made me understand the topic not memorize it with no understanding and forget it the next day at first i thought it's a pretty hard subject but your simplification of the topic with pictures showing us the bridges between the bacteria and the experiment that was done made me understand what's going on thank you so much please share more videos we need more people like you i'm an auditory person and love learning from watching videos you deserve a big like thx a lot again
Thank you for your kind words. I am happy you found them useful.
Finally all DNA level structures well explained! No others videos like this one.
I'm late to the genetics party, but thank you so much for this resource! I was having trouble figuring out what was going on with Hfr gene mapping, but you illustrated it so well. Much appreciation to you!
I'm glad you found it helpful!
which book are you using?
Concepts of Genetics by Brooker
Thanks Prof.Straceski !
You are welcome!
Very Good ! Thanks
Glad it helped!
If I have the length of a protein given as the numbers of aminoacids, can I say that the corrisponding mRNA has a minimum length of the number of the aminoacids times 3? Or does the UTRs also have a set minimum length?
UTRs contain at the minimum the Kozak sequence for ribosome binding at the 5' end and the AAUAAA cleavage and polyadenylation site at the 3' end, and then the subsequent poly A tail (usually). I 'm not aware of any measurements of average or minimum length, but I'm sure there are some!
Why doesn't the endonuklease cut just the tRNA out instead of cutting it with that extra bit of intron on it? Why the extra step with the exonuklease?
The “why” question is a tricky one. There is seldom a satisfying answer. The short answer is that there is no good explanation that has been elucidated right now.
@@janineleblanc-straceski3181 Hahaha fair enough! Thank you so much for your amazing lectures and also replying to all questions! I really appreciat it <3
why does the DNA only gets gut between histones? What prevents it from beeting cut at the curled around parts? Also Thank you for these awesome videos!
That is a great question. As DNA is replicated - or transcribed - nucleosomes dissociate from the DNA and reform behind the polymerase. There's a lot going on at the replication fork and the transcription bubble!
U r aweersssome
I’m glad you found these helpful.
Mam u r really aweeeeeeeeesssssoooommmmmeeee
Thanks!
U r really aweeeeeeeeeeessssssssooooooooommmmmmmmeeeeee
I love your course! Thank you for sharing! I just wish the music in the background was absent. This one is quite distracting.
Sorry about that.
I like the explanations
It very good Hello Dr LeBlanc Please 🙏 I need this unit 14B by book or PDF
The text is Concepts of Genetics by Brooker
@@janineleblanc-straceski3181 how to get it?
One of the most useful videos on this topic!
Thank you. I'm glad it helped.
Thank you for the posting these lectures. Have watched each one and found them to be very helpful.
I’m so glad you found them helpful!
very good explanation
Glad it helped!
Thanks for posting these vidoes, doc. Very helpful.
kitsand Thank you!
It means that the length of chromosome will become longer and longer and include much more useless information?
Chromosomes shorten overtime, at the telomeres.
@4:00 very good explanation about where and how to choose position to split the chromosome.
what a beautiful explanation :)
Thanks a lot for this playlist, its quick way to brush up the concepts. Is there any possibility to get these presentation slides from any of your sites? Thanks in advance.
Firstly thankyou mam , in first video lecture u told about ppts so can u provide link for that 😊
Best lecture on transposons on whole youtube
what are the protiens responsible for adding or putting the newly synthesized dna aroung histone is this occur at the same time when the new stard is just synthesized a histone protien in added?
That is a good question! There probably are some chaperone proteins. The newly synthesized DNA is immediately rewound around nucleosomes. Old nucleosomes are stripped off the template, broken down into half-nucleosomes that retain their epigenetic modifications (methylation, acetylation etc.) and mixed with brand new histones. Enzymes recognize the half- modified nucleosomes and duplicate the modifications on the new histones. (Re-call that a nucleosome core is an octamer of 2 each of H2A, H2B, H3 and H4.) That is how epigenetic modifications are passed from mother to daughter cells.
thank you so much. you are a saver
Very glad you found it helpfull.
@@janineleblanc-straceski3181 thank u sooo much
what signal the cell or tell the cell which region of dna it need to form a protien specific for the antigen what kind of signaling molecules involved here ofcourse the cell doesnt try all the possible 28 million outcome untill some one function xD
I have a question about the Ames test. Does the test only work with histidine revertants, or is it possible to use others?
The revertants are dependent on the mutation in the original strain of bacteria used. For example, Salmonella typhimurium has two different his- mutants that are used in the Ames test. Amino acid biosynthetic operons typically encode several enzymes that work in a pathway to synthesize the amino acid. Mutations in hisD and hisG genes have point or -1 frame shift mutations to enable the researcher to detect different mutagenic properties of a substance.
Thank you for posting. Surprised that PALB2 (which was almost named BRCA3 but is more like BRCA1.5 since it putatively joins BRCA 1&2 at some point) was not mentioned, especially since pathogenic mutations in that often results in cancer or prophylactic surgeries. Was looking for how it functions & any partial redundancies it may have w/ RAD 51 (C&D?) in conjunction w/ BRCA2 in HR/DSB repair...
I may add some details in a future version of the video. There is a good Wikipedia entry on it.
Your video is still helpful for many. Esp those working backwards from a genetic diagnosis.The clinical recommendations for people who hold DS repair gene pathogenic mutations are evolving (increasing) relatively rapidly for breast, ovarian, pancreatic (&?). The role of PARP inhibitors may also be an interesting upper level undergrad-ish level discussion, highlighting the functions of deleterious version of these genes. Even if students don't end up having big careers in medicine/biotech, having the ability to assimilate these concepts can add up to potentially life saving personal decisions. [read: finally, my education is paying off more than any science job I've had, LOL]
First, I really thank you for making this useful video. I watched this video from South Korea, and I'm highschool student interesting in brca gene. I know human use some methods when reparing DNA, and I thought that the one you mentioned in video is HR. Is it right? If it is right, if we use brca gene for dna reparing, does it always do its role with HR? Why it do not use NHEJ? If we have gene mutation either brca 1or brca 2, not both, is the possibility of cancer same?And what are other reasons causing dna mutation? If a person is inherited mutated gene, why breast cancer(or other else) happend in realtive old age, not in childhood?I'm sorry for my poor english...But I would really appreciate if you would leave some comments with my questions. Thank you!!
Very cool that you're studying this in high school. Was looking for words to respond to your question when I saw this in wiki: "When a double strand DNA lesion is repaired by NHEJ there is no validating DNA template present so it may result in a novel DNA strand formation with loss of information." That loss of information can still result in the production of a different protein or truncation of the protein that was supposed to have been produced. In individuals well past the embryonic stage [meaning anyone who can read this ;-) ], there are limitations to delivery of any repair mechanisms.