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Best is to measure soft plaque with CIMT

Wait for LMHR

so abundantly fun!

Been on the receiving end. Every box checked. Also add humiliation,isolation from friends and family, attempts to provoke physical response ( unsuccessful thankfully), attacks on property with weapon a hairline away from physical assault,psychotic and narcissistic behaviours, passive aggresion and attempts to justify all of it with insecurities,severe underappreciation of my high effort care. Almost three years to break free.Depression,anxiety,dangerous thoughts,addiction flared up. No more desire of having relationships. Very slow recovery. Not fun. Don't wait for all boxes to check,just one is more than enough to start looking for escape. Make yourself your priority.

Are you sure ApoB is atherogenic? How do you know

🍃🌱🍃🐛 👀

I want to learn whiteboard animation.how I can?

You know they said " don't use a lipid panel "because it measures only LDL and triglycerides. But then they are saying apoB measures all of the LDL's together, and since LDL is the higher number out of all of the LDL's it is the way to measure. Does not make sense, they are saying using apoB is basically the same as doing a Lipid panel, but adds in lipids that are not bad for you as part of the count. So sounds like another way to make your count high and justify....wait for it.....more statin's. Do a LIpoprotein anylysis NMR test, this will tell you all of it, count and sizes, look it up on line.

Wandering about in the woods of the LDL conspiracists. Come into the real world of the thrombogenic explanation. No big Pharma deception here. That’s five minutes of my life that I won’t get back 😱

I ALSO HATE DOODLE GOOD BYE AUFWIEDERSEHEN

"Statins stimulate atherosclerosis and heart failure: pharmacological mechanisms" Harumi Okuyama et al. Expert Rev Clin Pharmacol. 2015 Mar.

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CAC Scan is the best option. BTW, Cholesterol doesn¨t cause CAD. Inflamation is the cause.

Best treatment for atherosclerosis: Vitamin C Why do only coronary arteries clog with cholesterol and not veins or small capillaries? Scurvy = No vitamin C in your diet. Symptom: Your blood vessels break and you bleed to death. Think of the sailor of the past. Scurvy of the heart = Just enough vitamin C from food. Symptom: Arteries around the heart are not strong enough to resist high blood pressure. Damage is repaired with cholesterol LP(a) to prevent worse. After years of repair, your arteries become clogged. Enough vitamin C supplement of at least 3000 mg. per day gives strong and flexible arteries. Cholesterol is not needed as a repair agent. Cholesterol in your coronary arteries is broken down and burned in your liver. Source CZcams: - Cardiovascular disease and vitamin C (Dr. Rath Foundation) - Ending the Cardiovascular Epidemic by Natural Means - Dr. Matthias Rath

I’ve learned more in this comment section than 100 videos on this subject! Thanks to all of you! Absolutely amazing! Great video also. Much love and wishing EVERYONE excellent health. Cheers to a long and happy life ❤🎉

☯️🌏🕵🏻‍♂️♻️👣🌌

Is this an ad for Pfizer? Every "treaatment" seems to be "horse a fuck load of statins in to you and hope for the best"...

🎯 Key Takeaways for quick navigation: 00:00 *🚀 Introduction to explaining complicated concepts simply* - The challenge of explaining complex topics in an understandable manner. - Recognizing the cost of poor communication across various mediums. 01:02 *🎯 Tailoring the message to the audience's needs and interests* - Importance of adjusting communication to what the audience knows and cares about. - Using importance and benefits to grab and maintain attention. 01:43 *✨ Creating engaging moments and simplifying explanations* - Building explanations piece by piece and avoiding excessive details. - Engaging the audience with questions, storytelling, and comparisons to familiar concepts. 02:26 *📈 Enhancing understanding with benchmarks and visuals* - Using comparisons and visuals to deepen audience understanding and retention. 02:53 *🧠 Mastery and simplicity in communication* - Emphasizing the need for a deep understanding of the topic and the use of simple language. - The importance of avoiding jargon and acronyms for clearer explanations. Made with HARPA AI

Great video, thank you

You had my attention until you recommended statins and super statins.

How is ApoB is afected by Apo little a?

merci

Merci

Merci

Merci

I had to plead with my PCP to screen APOb. He finally agreed but said “I wouldn’t know what to do with it.”

Stop fear mongoring

Eudora does not exist What you talking

Finally, a straightforward health message exclusively on apoB, with a clear call to action to take action early. Measurement on LDL-c alone doesn't give the whole picture and is a poor man's surrogate for apoB. Here in the UK, a few years ago, standard lipid tests started to give a new measure, "non-HDL cholesterol", which for many people is a better proxy for apoB as it includes other atherogenic particles such as VLDL and IDL and for the majority of people is a reasonable measure if I understand this right. But - there is no free apoB test on the NHS, but I am considering getting one done privately to check whether I am in the significant minority of people whose LDL-c (and even non HDL-c) is "normal" but whose apoB is high. I totally agree with the message that the earlier the better - as build up of plaque in the arteries can start in young people and steadily accumulate over decades, leading to so-called "sudden unexplained heart attacks or strokes". So the message for me from this is: test apoB early and regularly and take measures to reduce it so it's "lower for longer". I have heard of the analogy with smoking risk where it is the accumulated exposure of "pack years" of cigarettes which matters, so "high apoB years" is what we should care about.

"I'd like to consult with you. How are animations in videos created? Could you guide me on this? Thank you."

你好，视频做的特别棒！我想请教一下，视频中的动画部分是如何做的？该怎么联系你呢？

Nice work🎉

BS 😂. Here's the true cause. What is modifying Apo-B100 for beginners? Inflammation and propagation of oxidation of LDL particles by industrial oils like canola, sunflower & corn oils are pure poison leading to chronic inflammation.

So have the serum spun, in centrifuge, and type the particles A1, A2, B3,4,5,6,7. A1 and A2 are healthy, buoyant, and B are small dense, sometimes glycated.

I’m going on with this process

This is okay where the diet is high in carbohydrates but for for someone in Ketosis? Probably not. Elevated HDL and LDL have been shown to extend longevity for those on low to no carb diet so in this case the ApoB would also be elevated however ApoB/ApoA1 would be very low indicating low risk. So understanding the patient as well as the science is important.

Is she a medical student. Or work. For pharm

Needs to have ct plaque score before any medication

You have to check other markers.

A much better measure than Apo B is the ratio of Apo B to Apo A1. Apo B can be large but if it's small in relation to Apo A1 it is not correct to assume elevated risk.

Dave Feldman's research into this precise subject is being released during the first week of December 2023 - it is going to be interesting to see whether the advice given in this video is correct. NOTE: I am not saying that the advice is incorrect but am waiting for the Feldman point-of-view.

It's also very important to test Lipo(a), which is determined genetically.

Regarding cardiovascular risk, we need to focus on *diet and lifestyle,* not LDL or ApoB. All animals have cholesterol. It is vital to all animal life processes. LDL cholesterol will not cause strokes, heart attacks, or atherosclerosis unless we have *metabolic dysfunction,* which occurs with chronic glycation, inflammation, oxidative stress, obesity, visceral fat, hypertension, hyperinsulinemia, insulin resistance, high triglycerides, and low HDL. We can avoid every one of these, and avoid strokes and cardiovascular disease, by avoiding fast food and processed food, and avoiding inflammatory seed oils (e.g. canola) and avoiding sugar, including starches (e.g. rice, bread, pasta & potatoes). Proper lifestyle means stress management, plus regular exercise. Big Pharma does not want you to think about diet and lifestyle. Pharma wants to sell you statins and PCSK9 inhibitors. Therefore pharma wants you to only focus on reducing LDL numbers, including ApoB numbers. *This is deadly.* No matter how low your LDL numbers become, you will have a stroke or heart attack if you have a poor diet and lifestyle. If you have metabolic dysfunction, then high LDL is a liability. On the other hand, if you have metabolic *health,* then high LDL is a *benefit.* You will live longer, with far fewer infections. Cholesterol is your friend until you make it your enemy. Don’t worry about cholesterol or ApoB. Focus on improving your diet and lifestyle.

Salient info given so well

To sell more statins. There are no long-term studies yet on what it says about ApoB

Excellent review on the importance of measuring ApoB for assessing CVD risk. I see that you mentioned the importance of Lp(a) and that you'd cover it in the next video. However, it's been almost a year and i still don't see a video on Lp(a). Any plans to discuss this contributory risk factor and how to include it when assessing CVD risk?

Its electronegative LDL and VLDL that is the real risk and these tests do not address that. Where is the research on electronegative LDL, there ARE papers but they are ignored.?

Thank you for a informative video on ApoB I am curious though you only list three pharmaceuticals as treatment options ...nothing about possibly better nutrition & exercise. Why is that?

Measuring ApoB is expensive and unnecessary. You want to know how much ApoB is DAMAGED and for that, there are already datapoints in your lab values: A1c and triglycerides/HDL. These two numbers should be low, the lower, the better. The higher the worse your cardiovescular risk. Why does this work? ApoB is a protein and Glucose attatches to it in roughly the same amount than hemoglobine. And when ApoB is attached with glucose, it becomes damaged. The liver does recycle LDL molecules with an undamaged ApoB. It refills them and sends them again on their way. If ApoB is damaged, the liver ignores it. These damaged LDL have to be recycled by macrophages. And where reside most of these? In the walls of the arteries. Here is the catch with LDL: it is not the total mass of LDL, which damages your arteries and your heart IT is the LDL molecules which have no functioning ApoB anymore. What we would like to know is the ratio of damaged LDL vs good LDL. And here comes triglycerides/HDL into play: it correlates highly with this ratio (about 0.9 which is as good as it gets, a correlation of 1 would be perfect). Get your fscking blood sugar down, eat healthy fats (not the plant based crap... put that into your Diesel engine). Do not take statins. At all! These do more harm than good.

OK…but I don’t think that there are any pharmacological agents that reduce Apo(b)…only Triglycerides, VLDL, IDL, LDL.